Equine

No Foot, No Horse

Published

5 days ago

May 22, 2025

By Dr. Garrett Metcalf, DVM

There is a wise old saying no foot no horse and that is absolutely true. Horses of all breed, discipline and size must have good healthy feet or they will suffer poor performance, chronic pain or worse succumb to diseases of the foot. There are several medical conditions that require surgical treatment within the hoof wall of the horse and this article will highlight the most common conditions that require surgical treatment and specialty farrier care.

Foot Abscesses –

Foot abscesses are a very common issue that nearly every horse may experience at some point in their lifetime. Abscesses are often minor issues that can be easily corrected by a farrier or veterinarian getting access to the abscess to allow drainage but they can be rather debilitating and sometimes rather serious. Abscesses in general are localized pockets of infection that found its way into the sole or white line of the foot. These abscesses often form because there is some structural abnormality of the foot, trauma that led to bleeding under the sole or improper hoof care that has led to abnormal forces being applied to the foot and of course the old hot nail. For example trimming of the foot without relieving enough sole pressure can lead to overloading the sole and in turn sole bruising setting up for an abscess. Other common abnormalities of the foot that leads to abscessation are laminitis and club feet. These two conditions can cause tearing and stretching of the white line and allow bacteria plus moisture to enter deeper into the foot which in some cases can further destabilize an already unhealthy foot, leading to a life threatening situation. Deep abscess that go untreated for days or weeks can continue to invade and dissect through tissue planes leading to larger abscesses. These large abscess sometimes require surgical intervention to keep them from spreading and to eliminate the abscess all together.

Pedal Bone Osteitis

Pedal bone or the coffin bone is a very unique bone compared to others in the horse. The coffin bone is a rather porous bone that has intimate attachment to the foot capsule and sole. The bone and the hoof tissue has a very high amount of blood supply rightly so because of the vast amount of metabolic rate energy it uses to keep the foot supplied with nutrients. Whenever the hoof is diseased or compromised from laminitis or infection the blood supply can be compromised as well spelling disaster. The disaster that can ensue from these conditions is an infected portion of the coffin bone or sequestration of bone. Bone sequestrums are when bone lacks blood supply and is also infected by bacteria that thrive off of dead tissue. Bone sequestrums are generally rather treatable conditions because once removed the bone can heal but the coffin bone is not the same as other bones in the horse. The coffin bone lacks an outer soft tissue coating called periosteum. Periosteum is a very robust membrane outside of almost all bones that provide blood supply and support healing with progenitor cells and stem cells. The uniqueness of the coffin bone without this important layer leads to poor healing, a more delicate blood supply and makes is more prone to infectious insults.

Treatment of an infected piece of the coffin bone requires aggressive steps in order to prevent spread and destruction of the rest of the coffin bone. Further spread into the coffin bone can lead to further damage to the blood supply to the bone and hoof as well as weakening the bone to the point of fracture under the weight of the horse. Aggressive surgical debridement or removal of infected tissue and bone is the first required step to reduce the amount of infection present in the foot. Secondly is aggressive antibiotic therapy using local delivery methods and systemic routes of administration. Local antibiotic delivery is by means of antibiotic beads, pastes or ointments and by means of regional limb perfusions. Regional limb perfusions are 20-30 minute treatments where antibiotics are delivered to the affected limb via blood vessels in that limb. The antibiotic is held in the limb by a tourniquet above the application site to allow higher concentration of the drug to enter the target tissue or region of the limb. Lastly is proper support of the remaining hoof while still maintaining access to the infected areas to allow local treatment. This step cannot be overlooked and requires the work of a talented farrier to make it possible.

Quittor

Quittor is a chronic deep infection within one of the collateral cartilages of the coffin bone. The collateral cartilages are attached on both wings of the coffin bone and are often referred to on x-ray films as side bone. Lacerations, puncture wounds, trauma and abscesses of the foot can lead to infection of the collateral cartilage. To most people quittor doesn’t sound like a big deal and seems like it would be easily addressed with a few days of antibiotics but that is not the case. This infection deep in the foot can be like a smoldering fire that cannot be put out until the infected cartilage is removed. The diagnosis is usually straight forward because there is often a draining tract with swelling, heat and proud flesh centered over one of the collateral cartilages. The difficulty lies in finding and removing all of the infected tissue not to mention that you have to go through the hoof wall to get there. A hoof wall resection or a window cut in the side of the foot is often needed to access the infected tissue, allow drainage and local treatment at the same time. Quittor can be rather difficult and sometimes require multiple surgeries in order to get the infection cleared up. After the hoof wall resection is made often a specialized shoe will be needed to help protect and keep the foot stable until the hoof grows out the defect in the hoof wall.

Keratoma

Keratoma is a benign tumor like growth that arises from the hoof wall or laminar tissue of the foot called keratin. Keratin is what makes up our hair and nails. This growth continues to expand between the foot wall and the coffin bone leading to pressure necrosis and damage to the coffin bone. This abnormal keratin tissue is usually located at the toe region of the foot and is thought to be triggered by trauma to the hoof tissue. The most common signs of a keratoma are reoccurring foot abscesses in the same location and same foot, plus lameness that are localized to the foot. X-ray, CT and MRI can be used to diagnose keratoma formation within the foot. Often the keratoma is well formed enough to be seen with x-ray but sometimes advance imaging is necessary to make the diagnosis.

The only treatment and cure for a keratoma is surgical removal through the hoof wall. This requires a hoof wall resection with either an oscillating saw or drill bit to removal the hoof wall without damaging the coffin bone. A keratoma has an often distinct appearance by this off white crumbly type tissue that is often easily removed from the surrounding healthy hoof wall. After surgical removal a specialized shoe is needed to protect the foot and allow access to treatment of the surgical site to prevent infection.

Coffin Bone Fractures –

There are many different patterns or ways that a coffin bone can be fracture and some are more serious than others. To keep it simpler we break them down into articular or non-articular meaning do they enter the coffin joint or do they not. Non-articular coffin joint fractures generally are much less serious and can be healed without major surgery. Often times non-articular fractures are stabilized with a special shoe and casting tape placed around the foot to make the hoof itself the “splint” for the coffin bone nestled inside the hoof wall.

Articular fractures of the coffin bone are a much more serious problem because of the damage that is done to the coffin joint. A fracture into any joint is a serious threat to the health of the joint and requires surgical reconstruction as soon as possible to keep the joint from developing crippling arthritis. The trouble again with any surgery on the foot is that the bone is inside the foot making it difficult to access. There are techniques to place screws into the coffin bone through small hoof wall resections to allow stabilization of coffin bone fractures. It does require the right fracture pattern and location to make this surgical treatment a plausible option.

Street Nail

A street nail surgery is used to treatment of deep penetrating injuries that occur at the frog or sole that leads to infection of the bottom of the coffin bone, navicular bone and closely related surrounding soft tissue structures. Street nail procedures are often needed when a metal object such as a nail or screw penetrates and infects the one of the vital structures of the bottom of the foot. This window allows flushing of the synovial structures and removal of damaged/infected tissue. This procedure success is greatly improved with the use of an arthroscopic camera placed in the navicular bursa or coffin joint depending on what area the puncture wound involves. The arthroscope allows better visualization and more thorough flushing of debris and infection out of these tight spaces. Again this surgery cannot be successful with the application of a special shoed called a hospital treatment plate shoe that allows access to the bottom of the foot while keeping the foot clean and protected.

As you can see there is a pretty clear pattern to these hoof conditions: infection and the need for specialized farrier care. In order to be successful in treating these conditions the veterinary surgeon and farrier must work hand in hand to provide the best care for the horse. Although performing surgery on the foot of a horse is challenging and sometimes limited, it is often possible to have successful outcomes with a variety of different conditions.

Equine

Horse Pasture ManagementBy OKFR Staff

Published

4 days ago

May 23, 2025

bowienews

As spring weather arrives, horse owners are often eager to turn their horses out to graze and exercise—but is the pasture truly ready?

With proper management, pastures can serve as a high-quality forage source and a safe space for exercise. Effective management can even extend the forage production season, helping to reduce hay expenses. On the other hand, unmanaged pastures often develop weed problems, lack nutritional value, and can pose health risks for horses.

Here are five key steps to help improve and maintain horse pastures:

1. Soil Testing, Fertilizing, and Liming

Annual soil testing is ideal, but due to cost, testing every five years for grazing pastures and every three years for hay fields is a reasonable alternative. Grazing pastures tend to lose nutrients more slowly than hay fields because some nutrients are returned to the soil through horse waste, whereas nutrients in harvested hay are removed entirely.

For guidance on soil testing, fertilization, and liming, producers can refer to the Oklahoma Forage and Pasture Fertility Guide, available through Oklahoma Cooperative Extension and OSU’s Division of Agricultural Sciences and Natural Resources.

2. Over-Seeding and Renovating Bare Spots

Over-seeding helps replenish forage stands and prevent weed invasion. Use a high-quality pasture seed mix, limiting legume content to no more than 25 percent to avoid excessive salivation in horses.

3. Sacrifice Area

Creating a sacrifice area—a small, fenced section (approximately 300 square feet per horse)—gives overgrazed pastures time to recover during periods of heavy grazing or adverse weather. Horses kept in sacrifice areas should always have access to clean water, adequate hay, good drainage, and waste management.

4. Rotational Grazing

Horses are more selective grazers than cattle and will often overgraze their preferred plants, eventually killing them. This leads to spot grazing and pasture degradation. Rotational grazing allows plants time to regrow and helps reduce waste due to trampling.

Manure concentration in preferred areas also decreases grazing efficiency and nutrient distribution, making rotation an essential tool. Horses should be moved to a new pasture when forage is grazed down to 3–4 inches in height.

5. Weed Control

Chemical herbicides are commonly used to manage weeds, but identifying the specific weeds present is essential for selecting the right treatment. To prevent herbicide resistance, avoid long-term use of the same active ingredient and consider complementary methods such as mowing.

Maintaining healthy pasture also includes proper fertilization and rotational grazing, which both help minimize weed invasion.

Forage Selection and Toxic Plant Awareness

Selecting the right forage species depends on factors like land resources, management goals, and available capital. A mix of cool- and warm-season forages generally provides the most cost-effective year-round grazing.

Warm-season grasses, such as Bermuda grass, are commonly used for their high yield, good nutritional value, durability, and responsiveness to nitrogen fertilization. Alternatives include old world bluestem and crabgrass.

Alfalfa, a warm-season perennial legume, is widely used as hay in Oklahoma and can be grazed, though grazing may not always be economical.

Cool-season grasses such as wheat, rye, oats, barley, and ryegrass can be sod-seeded into dormant warm-season pastures to extend the grazing season. Combining ryegrass with small grains often allows grazing later into the spring. Cool-season clovers like red, white, and rose clover also thrive in Oklahoma and provide excellent nutrition.

However, it’s important to recognize and remove potentially toxic plants from horse pastures. These include bitterweed, black locust, cocklebur, horsetail, milkweed, ornamental yew, pigweed, snakeroot, St. John’s wort, wild parsley or carrot, and yarrow. Grasses in the Sorghum genus can contain prussic acid, which may cause severe health issues and even death. Clovers and small grains in humid conditions may also develop mold, which can be dangerous to horses.

Equine

Grain Overload in Horses

Published

3 months ago

March 7, 2025

bowienews

By Dr. Devon England

You come home after a long day at work, and your feed room door is wide open and grain bins are empty. What do you do? Before you call your vet, figure out what grains were eaten, try to estimate how much and determine which horse or horses got into the feed. Did your horse just get into some hay or alfalfa pellets, or did he get into a high sugar grain like Omolene 300? Did your horse get into chicken or cattle feed? If he did, is it a medicated feed, containing an ionophore like monensin or lasalocid?

Grain overload is an emergency, with better outcomes, the sooner the horse is treated. So, call your veterinarian as soon as you figure out a possible grain overload has occurred. Based on what feed and how much feed your horse consumed, your veterinarian can try to determine the risks to your horse and come up with a treatment plan. Signs of grain overload include colic, lethargy, inappetence, diarrhea and lameness. These signs will vary depending on how long ago grain ingestion occurred and the severity of grain overload.

If your horse got into hay or alfalfa pellets, these are low in sugar and therefore not a high risk for problems. Your horse may just have a mild belly ache (colic) episode. If your horse got into a feed like Omolene 300, they are at risk for grain overload. How severely they are affected will depend on a few factors including how much grain was eaten (5lb vs 30lb), if the horse normally receives that grain or doesn’t receive any grain, and the size of the horse vs the amount of grain eaten. Eating 5lb of grain for a full-size horse who normally receives grain may not be a problem, but that same amount of grain eaten by a small pony who normally only receives hay will be a problem.

Treatment for grain overload should be initiated as soon as possible. This includes lavaging the stomach to remove any remaining grain. Stomach lavage is only successful in removing feed within the first couple of hours. Horses will be given mineral oil or charcoal to decrease absorption of breakdown products (acids) from grain fermentation and protect the intestinal lining from damage. Intravenous fluids are given, because horses can quickly become dehydrated with grain overload, due to digestion products from the grain drawing in a lot of water into the colon. Anti-inflammatories, lidocaine, and ice boots are used in more severe cases to decrease the risk of laminitis. In more severe cases with damage of the intestinal lining, antibiotics may be used to prevent infections associated with bacterial escape from the damaged intestine.

If your horse got into medicated feed containing an ionophore, your horse is at high risk for heart failure and muscle damage, on top of grain overload. Ionophores are toxic at very low doses in horses (2mg/kg). These drugs are added to livestock feed to decrease parasites (coccidia), to help with feed efficiency, to decrease risk of bloat and to decrease methane emissions. If your horse has eaten ANY amount of medicated feed, they should be immediately seen by your veterinarian, even if they are not showing any clinical signs. Horses can have no clinical signs or be mildly lethargic in cases where small amounts of medicated feed were eaten but still have evidence on blood work or muscle and heart damage. Clinical signs include initial inappetence or colic signs, weakness or neurologic signs in the hind end, muscle fasciculations (twitching), laying down more, and not being able to stand. There is no anti-dote to ionophore toxicity. Treatment may include mineral oil, IV fluids, anti-inflammatories and supportive care, depending on how soon toxicity is identified. In severe cases, horses can die or develop heart failure, necessitating euthanasia. In less severe cases, especially if identified and treated early, horses can recover fully.

Grain overload can come with serious complications if severe or medicated feeds are involved, especially if treatment is delayed. Secure your feed room as best as possible and keep medicated livestock feeds in a separate location to prevent cross-contamination. And if you have any concerns about your horse getting into feed, call your veterinarian as soon as possible.

This article was originally published in the February 2025 issue of Oklahoma Farm & Ranch.

Equine

Gastric Ulcers

Published

4 months ago

January 20, 2025

bowienews

By Dr. Devan England DVM

Does your horse have gastric ulcers? Gastric or stomach ulcers are frequently blamed for a variety of things including poor performance, acting ‘cinchy’, weight loss, not eating, poor coat condition, diarrhea and colic. However, gastric ulcers are not always the culprit and the only way to know for sure if your horse has gastric ulcers is to look at the stomach on camera, using an endoscope. Poor appetite and poor body condition are the mostly widely observed clinical signs with gastric ulcers, however, these are non-specific. If you think your horse might have gastric ulcers, the best place to start is to talk to your veterinarian and consider scheduling a gastroscopy. Gastroscopy requires the horse be held off feed for at least 16-18 hours and held off water for at least 6-8 hours. Fasting off feed and water is necessary to allow the veterinarian to see the whole stomach. If restricting feed or water is difficult in your management situation, many veterinarians will allow you to hospitalize your horse the night before gastroscopy for proper fasting.

Gastric ulcers are split into two types, classified by the location of the ulcer in the stomach. Squamous ulcers are ulcers that occur in the squamous or skin like portion of the stomach. This is the top part of the horse’s stomach, is closest to the esophagus, and has squamous tissue to protect this portion of the stomach from stomach acids. The other ulcer type are glandular ulcers. Glandular ulcers occur in the bottom portion of the stomach, which is closest to the small intestine. This portion of the stomach has glandular mucosa with cells responsible for producing stomach acids for digestion as well as cells that produce mucus and buffers to protect the lining from stomach acid. Gastroscopy is important not only for diagnosing whether ulcers are present but also determining the severity and the type of ulcer, because these two ulcer types require different treatments.

Squamous gastric ulcers are common in racehorses both in and out of training, with higher prevalence in racehorses under training. Prevalence in Thoroughbred racehorses in training has been reported to be up to 100% (Sykes 2015). Squamous ulcers are also prevalent in Western pleasure horses, Thoroughbred stallions on breeding farms, and Italian donkeys (Sykes 2015). Glandular gastric ulcer prevalence has not been as well described as squamous ulcers. Glandular ulcers are reported to be most common in Thoroughbred and Standardbred racehorses, Canadian showjumpers and polo ponies, and American Quarter Horses (Sykes 2015).

Risk factors for ulcers vary by ulcer type. Anti-inflammatories (Bute, Banamine) can increase the risk of glandular ulcers in some horses by affecting normal defense mechanisms but are not a high risk in most horses. Horses that display stereotypic behaviors, such as cribbing, have an increased risk of squamous ulcers. Grain fed before hay in non-exercising horses, feeding larger amounts of grain, and increased time between meals increases the risk of squamous ulcers. Increased time with high intensity exercise and housing in single pens is associated with increased risk of glandular ulcers. A straw only diet, lack of water access and lack of direct contact with other horses increases the general risk of gastric ulcers.

If your horse is diagnosed with ulcers, the mainstay of treatment is a buffered formulation of omeprazole (Gastrogard, Ulcergard). Over the counter Omeprazole and compounded Omeprazole are not effective because without buffering, the acidic stomach quickly breaks down the drug before absorption. Most horses with squamous ulcers will have healing of these ulcers after a 4-week course of Gastrogard or Ulcergard at treatment dose (whole tube for the average horse). Some horses may be healed by 3 weeks of treatment, but all horses should undergo a recheck gastroscopy before stopping treatment. Horses diagnosed with glandular ulcers need combination therapy with Gastrogard/Ulcergard and Sucralfate for 4 weeks. About 2/3 of horses with glandular ulcers will heal in this time, but some horses may require longer treatment times so a recheck is always recommended before discontinuing treatment.

Horses at higher risk of gastric ulcers may benefit from preventative (low) doses of Ulcergard (1/4 tube in average sized horse) given for a few days before and during high stress situations like long distance travel and competitions. Sea buckthorn berry supplement may be protective against formation of glandular ulcers. Dietary management to decrease the risk of ulcers includes providing more frequent small hay meals if pasture access is not available, limiting high sugar grains as much as possible and adding vegetable oil to the feed.

Sykes BW, Hewetson M, Hepburn RJ, Luthersson N, Tamzali Y. European college of equine internal medicine consensus statement – equine gastric ulcer syndrome in adult horses. J Vet Internal Med 2015; 29:1288-1299.