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Equine

Umbilical Hernia

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By Lauren Lamb

An umbilical hernia is a defect in the horse’s ventral body wall at the location of the umbilicus. The umbilicus is the equivalent of our belly button. The umbilicus is where the umbilical cord attaches to the foal while it is developing in the mare’s uterus. The umbilical cord is made up of a vein, two arteries and the urachus. The umbilical vein and artery carry blood to and from the fetus as it is developing in the uterus. The urachus is a tube that connects to the fetus’ bladder and carries urine from the developing fetus.

Umbilical hernias are seen in foals between the ages of birth and three months of age. The hernia bump seen on the bottom of the abdomen is made up of skin, subcutaneous tissue, peritoneum (lining of the abdomen) and a piece of the gastrointestinal tract.

Questions concerning umbilical hernias frequently asked by owners:

  • How does an umbilical hernia develop?
  • Are they genetic?
  • How can I prevent an umbilical hernia?

Some evidence shows umbilical hernias may have a genetic component; however, the genetic influence is most likely mild. This means breeding a mare and/or stud that had an umbilical hernia when they were foals will not result in an offspring with an umbilical hernia. The flip side of the coin is also true; crossing a mare and stud with no history of umbilical hernias will not result in a foal without an umbilical hernia.

Some have a strong belief that foaling behavior of the mare immediately after delivery can have an influence on whether a foal will develop an umbilical hernia. The natural behavior of a mare following delivery of her foal is to remain lying down until the foal starts to move and becomes more active. This may take several minutes for the foal to become active. Once the foal becomes active, the mare will stand up and break the umbilical cord about one inch from the foal’s navel. When a mare stands prematurely, the risk of an umbilical hernia forming can be increased. The most common reason for a mare to stand prematurely is human intervention. When humans try to rush into the stall to assist the foal or mare, the mare will become nervous and stand up too early.

An umbilical infection can lead to an umbilical hernia. The post foaling care of the umbilicus can influence whether or not a foal will develop an umbilical infection. The umbilicus should be dipped in dilute Nolvasan solution or betadine solution two to three times a day for the first three days of life. An umbilical infection is likely to occur if the umbilicus is not dipped in an antiseptic solution for the first three days after birth.

The size of an umbilical hernia is measured in fingers, aka the number of fingers that will fit in the hernia. A small hernia is less than three fingers in size. A large hernia is anything bigger than three fingers in size. The hernia should not be painful on palpation, nor should there be any heat or swelling around the hernia. When you place your fingers in the hernia, the contents of the hernia should be easily reduced (pushed back into the abdomen). A veterinarian should be contacted as soon as possible if you notice any heat, swelling, pain on palpation or you cannot reduce the hernia. A veterinarian should also be contacted as soon as possible if a horse with an umbilical hernia starts to colic. All these clinical signs are evidence that a portion of the intestine is stuck within the hernia. When the intestine is stuck within the hernia, the blood supply to the intestine can be compromised, which will result in the intestine becoming necrotic—dead. The hernia should be checked daily to ensure none of the previously mentioned clinical signs have developed.

Several treatment options are available for hernias. One conservative treatment option for small hernias (less than three fingers), is to reduce the hernia one to three times a day. By reducing the hernia you allow the hernia ring (defect in the body wall) to heal close. When the foal reaches four months of age, there is little chance that the hernia will heal with conservative therapy. Any foal that is greater than four months of age with an umbilical hernia will need a surgery to repair the hernia.

An umbilical hernia has two surgical repair options—both require general anesthesia and can be performed in a clean stall or a surgery room. A hernia repair performed in a surgery room has lower complication rate following surgery.

The first surgical option is called the closed technique. With the closed technique, the abdominal cavity is not opened, but the skin and subcutaneous tissue are opened. The hernia is repaired by placing large sutures across the abdominal defect. The closed technique decreases the risk of an infection occurring within the abdomen, but it increases the risk that a portion of the intestine is sutured to the body wall. The closed technique is usually used in cases were the surgery is performed in a stall within the barn. In this location, there is higher environmental contamination compared to a surgery room.

The open technique is the second option. With the open technique the abdominal cavity is opened and large sutures are placed across the defect to close the hernia. With the open technique, the risk of the intestine being sutured to the abdominal wall are significantly decreased. The open technique should be performed in a surgery room.

Following surgery, foals are kept on stall rest with no hand walking for two weeks and then turnout in a small paddock or trap for an additional two weeks. After four weeks, foals are allowed to go back to regular turnout and exercise.

Umbilical hernias are abdominal wall defects that are commonly seen in foals. The cause of an umbilical hernia is not completely understood; however, both genetics and husbandry practice can contribute to a horse developing an umbilical hernia. Surgery is not always required to repair umbilical hernias unless the foal is over four months of age. At this age surgery is needed to repair the hernia. Following surgery, foals had a good prognosis.

Equine

Degenerative Suspensory Ligament Desmitis of the Horse

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By Dr. Garrett Metcalf

The suspensory ligament is a vital component of the limb of a horse to produce normal locomotion and support. The suspensory ligament is a common area of concern in performance horses of various disciplines and can be single handedly the cause of lameness or performance issues. This article is going to look at a specific degenerative disease of the suspensory ligament and what horses are at risk for this disease.

DSLD or degenerative suspensory ligament desmitis was first discovered in the early 1980’s in Peruvian Paso horses. The name has been changed because the suspensory ligament is not the only organ affected from the disease but the suspensory is ultimately the biggest issue. The newer name, ESPA or equine systemic proteoglycan accumulation, is more correct because other ligaments and tissues are affected by this disease. In this article we will only focus on the suspensory ligament. The most commonly affected breeds are Peruvian Paso, Paso Fino, Morgan, Saddlebred, Warmblood, Paints, American Quarter Horse, and Thoroughbred breeds. The age of onset of the disease is variable among breeds but it is more common to be seen in middle age to older horses.  However it has been documented in horses as young as one year of age. The disease generally will have a slow insidious onset that can go undiagnosed for months or years depending on the horses work and discipline.

A horse that begins to show early signs of DSLD may have a vague lameness issue that is difficult to isolate and they most likely will resolve with a period of rest. As the horse returns to moderate level of work the lameness will return. This scenario may go on for several months or more before the discovery of the DSLD is made. The first indication of DSLD is often pain isolated in the suspensory branches or fetlock region when a flexion test is performed. Horses with DSLD will also have a “dropped” fetlock appearance because the suspensory is the main supporting structure of the fetlock joint.  DSLD can affect the hind limbs, forelimbs or all limbs at the same time. A unique sign of DSLD is that not just one limb is affected but rather bilaterally affecting the limbs, meaning it will either affect either both forelimbs or hind limbs at the same time. It is my experience that the hind limbs are more commonly affected compared to the forelimbs. Horses will often have enlargement of the fetlock region and increased joint fluid or wind puffs. Horses will often have a straight hock or post legged hind limb appearance. Horses will often shift weight frequently in an effort to get relief from the discomfort and this can be confused with other lameness issues or foot related pain.

Diagnosis of DSLD is often made by clinical signs, breed and ultrasound findings. Ultrasound imaging of the suspensory ligaments will often show diffuse enlargement of the suspensory body and branches. The suspensory ligament will have a poor heterogeneous fiber pattern with periligamentious soft issue thickening from scar tissue deposition and edema or fluid within the tissue.  Radiographs of the lower limb may reveal abnormal bone changes in the sesamoid bones behind the fetlock joints and even osteoarthritis of the pastern and or fetlock joints. A definitive diagnosis can be made from a biopsy of a ligament in the neck called the nuchal ligament, but is not often performed because of the invasiveness of the biopsy.

Treatment is very limited and it is mostly geared towards protection of further damage by prolonged rest. Pain management is also important to attempt to keep the horse as comfortable as possible. Different shoeing techniques can be used with marginal success. In early cases of DSLD, a suspensory shoe that helps engage more work from the deep digital flexor tendon can help elevate the fetlock and offer more protection to the suspensory ligament. The devastating thing about this disease is that there is no cure and there are hardly any good options to slow the progression of the disease. DSLD carries a poor prognosis when the diagnosis is made in any breed of horse or any discipline. Although some cases can be managed better than others, it often progresses to the point of debilitating pain and discomfort to the point of humane euthanasia especially in the Peruvian Paso breed.

Read more in the February 2023 issue of Oklahoma Farm & Ranch.

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Equine

Gastric Ulcers

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By Dr. Devan England DVM

Does your horse have gastric ulcers? Gastric or stomach ulcers are frequently blamed for a variety of things including poor performance, acting ‘cinchy’, weight loss, not eating, poor coat condition, diarrhea and colic. However, gastric ulcers are not always the culprit and the only way to know for sure if your horse has gastric ulcers is to look at the stomach on camera, using an endoscope. Poor appetite and poor body condition are the mostly widely observed clinical signs with gastric ulcers, however, these are non-specific. If you think your horse might have gastric ulcers, the best place to start is to talk to your veterinarian and consider scheduling a gastroscopy. Gastroscopy requires the horse be held off feed for at least 16-18 hours and held off water for at least 6-8 hours. Fasting off feed and water is necessary to allow the veterinarian to see the whole stomach. If restricting feed or water is difficult in your management situation, many veterinarians will allow you to hospitalize your horse the night before gastroscopy for proper fasting.

Gastric ulcers are split into two types, classified by the location of the ulcer in the stomach. Squamous ulcers are ulcers that occur in the squamous or skin like portion of the stomach. This is the top part of the horse’s stomach, is closest to the esophagus, and has squamous tissue to protect this portion of the stomach from stomach acids. The other ulcer type are glandular ulcers. Glandular ulcers occur in the bottom portion of the stomach, which is closest to the small intestine. This portion of the stomach has glandular mucosa with cells responsible for producing stomach acids for digestion as well as cells that produce mucus and buffers to protect the lining from stomach acid. Gastroscopy is important not only for diagnosing whether ulcers are present but also determining the severity and the type of ulcer, because these two ulcer types require different treatments.

Squamous gastric ulcers are common in racehorses both in and out of training, with higher prevalence in racehorses under training. Prevalence in Thoroughbred racehorses in training has been reported to be up to 100% (Sykes 2015). Squamous ulcers are also prevalent in Western pleasure horses, Thoroughbred stallions on breeding farms, and Italian donkeys (Sykes 2015). Glandular gastric ulcer prevalence has not been as well described as squamous ulcers. Glandular ulcers are reported to be most common in Thoroughbred and Standardbred racehorses, Canadian showjumpers and polo ponies, and American Quarter Horses (Sykes 2015).

Risk factors for ulcers vary by ulcer type. Anti-inflammatories (Bute, Banamine) can increase the risk of glandular ulcers in some horses by affecting normal defense mechanisms but are not a high risk in most horses. Horses that display stereotypic behaviors, such as cribbing, have an increased risk of squamous ulcers. Grain fed before hay in non-exercising horses, feeding larger amounts of grain, and increased time between meals increases the risk of squamous ulcers. Increased time with high intensity exercise and housing in single pens is associated with increased risk of glandular ulcers. A straw only diet, lack of water access and lack of direct contact with other horses increases the general risk of gastric ulcers.

If your horse is diagnosed with ulcers, the mainstay of treatment is a buffered formulation of omeprazole (Gastrogard, Ulcergard). Over the counter Omeprazole and compounded Omeprazole are not effective because without buffering, the acidic stomach quickly breaks down the drug before absorption. Most horses with squamous ulcers will have healing of these ulcers after a 4-week course of Gastrogard or Ulcergard at treatment dose (whole tube for the average horse). Some horses may be healed by 3 weeks of treatment, but all horses should undergo a recheck gastroscopy before stopping treatment. Horses diagnosed with glandular ulcers need combination therapy with Gastrogard/Ulcergard and Sucralfate for 4 weeks. About 2/3 of horses with glandular ulcers will heal in this time, but some horses may require longer treatment times so a recheck is always recommended before discontinuing treatment.

Horses at higher risk of gastric ulcers may benefit from preventative (low) doses of Ulcergard (1/4 tube in average sized horse) given for a few days before and during high stress situations like long distance travel and competitions. Sea buckthorn berry supplement may be protective against formation of glandular ulcers. Dietary management to decrease the risk of ulcers includes providing more frequent small hay meals if pasture access is not available, limiting high sugar grains as much as possible and adding vegetable oil to the feed.

Sykes BW, Hewetson M, Hepburn RJ, Luthersson N, Tamzali Y. European college of equine internal medicine consensus statement – equine gastric ulcer syndrome in adult horses. J Vet Internal Med 2015; 29:1288-1299.

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Equine

Trailer Safety Checklist

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By Janis Blackwell

As the season arrives to gear up for participation in your equine event of choice, one thing remains a constant for all horse owners. That constant is our responsibility to insure the safety of our horses by being diligent to maintain the integrity of the trailers in which we haul them. There are a number of things that can be dangerous both inside and outside of your trailer. Whether you traveled all winter long or whether your trailer sat unused or was used very little through the cold weather months, at least once a year your trailer is due a thorough going over. So here we go with a checklist that will help you insure a happy and safe trip for you and your equine partner.

  1. A sound floor is absolutely imperative. Whether your floor is aluminum, steel or wood, it should be cleaned regularly after use to preserve it. Urine and manure will erode and weaken all types of floors. Even rubber mats will not prevent erosion of your floor. (Maintenance tip: remove mats and wash aluminum floors often to prevent erosion.)
  2. Especially check wooden floors for rotten boards. Immediately replace questionable flooring before hauling. (Maintenance tip: For wood, remove mats and wash out manure and debris. Coat wooden floor in a cheap motor oil. Allow to sit in hot summer weather until the oil soaks in. Be careful—floor will be slippery until oil cures into the wood. This treatment yearly will preserve a wooden floor for much longer than normal as it repels urine and protects the wood).
  1. Keep the interior and exterior washed to enable you to check for rusted out places, leaks, etc.
  2. Have a professional check your brakes at least once yearly to be sure they are operating properly.
  3. Be sure tires are inflated to the proper air pressure, and check the inside of each tire for hidden unusual wear that could cause a blowout. Replace worn tires before leaving home.
  4. Wheel bearings must be checked and packed at least once a year. This should be done even if the trailer has been rarely used since the last time the wheel bearings were packed. In fact, trailer maintenance professionals say that sitting stationary and unused is even worse for the bearings.  Improper care and maintenance of wheel bearings can cause a wheel to seize up and actually twist off while in use. Use a horse trailer professional for this maintenance task.
  5. Axles should be checked for bowing. A bent or bowed axle can cause excessive tire wear and damage wheel bearings.
  6. There should be no more than two inches in height difference from the front of the trailer to the back. More difference than that causes the bulk of weight of the trailer and its contents to ride mostly on the rear axle causing it to bow and wear on both tires and wheel bearings.
  7. Another critical part of the trailer to keep an eye on are the butt chain or bar and the back door. The butt chain or bar should be firmly attached to the wall and its keeper and should always be latched. The door should have a strong secure latch with a pin to insure it stays latched while in motion.
  8. Finally, but certainly not of least importance is a thorough check of the trailer hitch including ball and coupling. Keep the ball well greased. Periodically, check to see that the ball is still securely tightened and the latch on the coupling is working properly.

These few critical safety check points can save you money, stress and the wellbeing of your horse. Until next time, happy trails and safe traveling.

This article was originally published in the April 2016 issue of Oklahoma Farm & Ranch. 

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