Equine
Core Vaccines Every Horse Needs
There are many possible dreadful diseases that horses can contract through various insect vectors, ticks, wildlife and even wounds that are rather easy to prevent with routine core vaccinations. Oklahoma has just about every insect known to man to offer, as all Oklahomans are fully aware of especially in the spring and summer, making it a very good environment for some of these key viruses to spread. The five types of diseases that will be discussed in this article are commonly referred to as the deadly five, and that is why they make the core vaccine list because if one of these diseases is contracted it can be game over for your horse. Before spring arrives, the core vaccines should be on every horse owners to do list as a common health care checklist that should be just as routine as deworming and dental care.
The American Association of Equine Practitioners (AAEP) and American Veterinary Medical Association (AVMA) have established key guidelines for vaccinations for horses that are required based on risk to the public and to the horse themselves. The five diseases listed as core vaccines are Rabies, Tetanus, Eastern equine encephalomyelitis virus, Western equine encephalomyelitis virus and West Nile virus.
The AVMA defines core vaccinations as those “that protect from diseases that are endemic to a region, those with potential public health significance, required by law, virulent/highly infectious, and/or those posing a risk of severe disease. Core vaccines have clearly demonstrated efficacy and safety, and thus exhibit a high enough level of patient benefit and low enough level of risk to justify their use in the majority of patients.” (AAEP website)
The reason these deadly five diseases are listed as core vaccines is because they pose a possible public health risk, are highly virulent or infectious, have a high mortality rate and can be effectively controlled with routine vaccination with great efficacy. It’s important to remember that vaccines are not 100% effective for every disease but well worth that risk when it comes to protect against these five deadly diseases.
There are many other diseases that horses may or may not need to be protected against with vaccinations. These diseases are listed under “risk based” immunizations depending on travel, environment, geographic circumstances and even competition/racing venue requirements before entering into an event. With recent localized outbreaks of equine herpes virus at race tracks, shows and barrel races, many venues and race tracks require very recent vaccinations for these viruses before permitted onto the grounds. Here is a list of risked based diseases vaccinations are available for: Anthrax, botulism, equine herpes virus 1 & 4, equine viral arteritis, influenza, rotaviral diarrhea and strangles. These diseases and the risk of contracting them must be weighed on an individual horse bases versus the risk of the vaccine itself.
Many vaccines today that are available for horses have all core vaccines in one shot except rabies. One vaccine provider has developed the first all five core diseases in one single vaccine. Horses just like other species require multiple boosters to generate an appropriate immune response to be protective. Unvaccinated horses or horses that don’t have a documented vaccine history it is recommended to have at least one booster four to six weeks after the first dose to get an adequate response and then annually after that. In some endemic areas of the country with higher cases of West Nile and other viral encephalomyelitis diseases due to prolonged mosquito vector season it is often recommended to have a booster in the mid-to-late summer to have added protection against these particular viruses.
Below is a breakdown of each disease and its importance for your horse and even yourself to be protected against.
Rabies –
Highly deadly virus that is unsurvivable for any mammal that contracts the disease and begins to show clinical signs, even for humans. As of 2016 only four people have survived rabies infection after showing clinical signs, so the odds are very poor. One of the most difficult aspects of rabies in horses is the variability of clinical signs. Horses can display two clinical manifestations of the disease: Furious/Mad form or Dumb/Stuporous but really it can look like anything early on. Veterinarians are trained that rabies can display almost as anything and should always be on the list for a horse that presents with neurologic symptoms, especially horses with no vaccine history for rabies. Horses can display colic, ataxia, blindness, hypersalivation, depression, weakness and even lameness. Also there is no ante-mortem test meaning there is no test to confirm rabies until the horse is dead or euthanized and the brain is collected for testing. Rabies is transmitted via bites from infected rabid wildlife such as skunks (highest transmitter in Oklahoma), raccoons, fox and bats.
EEE/WEE –
Eastern and Western equine encephalomyelitis is transmitted like other viral enchephalitides via the mosquito vector. Clinical signs are neurologic signs of ataxia, blindness, head pressing or head tilt, recumbency, seizures and paralysis of the throat/tongue. Only up to 15% of horses survive EEE. WEE has a better survival rate up to 50% and has very similar sign as EEE. Birds are common carriers of EEE/WEE and are the reservoir for these viruses.
West Nile –
A very commonly know virus that is also transmitted by mosquitoes to horses, humans, birds and other mammals. Birds again are the largest reservoir for this virus. The virus affects the central nervous system by creating inflammation around the brain and spinal cord. Symptoms or clinical signs are fever, depression, muscle fasciculations, weakness and ataxia. The severity of the disease is horse dependent and not all horses get the disease but among those horses that do about 1/3 will die from it. Also almost half of horses will have lasting neurologic effects from the disease.
Tetanus –
The causative bacterium was first discovered in 1884 by injecting wound exudates from humans with tetanus into animals leading to the discovery that Clostridium tetani is the causative bacteria. The bacteria is found everywhere in the environment including the soil and manure of horses. Horses are rather sensitive to the neurotoxin produced from Clostridium tetani that leads to the clinical signs of tetanus. The bacteria enter the horse’s body through wounds, cuts or even surgical wounds such as castration sites. The spores of the bacteria turn into the vegetative form when it begins to grow and replicate. The neurotoxin produced from the bacteria enters the central nervous system by peripheral nerves. The clinical signs of tetanus are muscle spasms, stiffness, prolapsed third eyelids, extended head and neck, elevated tail and retracted lips. As the disease progresses, horses will become recumbent and have difficulty breathing. Life saving treatments can be implemented to save the infected horses but still up to 75% of horses will die.
What all these disease have in common besides death is that these deaths are extremely excruciating, painful and miserable. There is no doubt from seeing these cases personally and from watching teaching videos that these horses are suffering enormously. What makes it even more difficult and disheartening when we see these cases is that they are so easily preventable with vaccination. The bottom line is that vaccines can save your horse’s life from these deadly diseases and vaccines are rather inexpensive especially compared to the expensive of trying to treat the horse once they are ill. Please talk to your veterinarian about getting your horse or horses protected with core vaccinations and if any other vaccinations are necessary based on your situation.
Read more great articles in the March 2020 issue of Oklahoma Farm & Ranch.
Equine
What’s That Test For?
By Ann Asher
For many horse owners, a Coggins test is part of the routine. If you’re headed to a horse show, rodeo, sale, boarding facility or across state lines, you have likely been asked for a current negative Coggins. The veterinarian draws a tube of blood, fills out the paperwork and, assuming the result comes back negative, the form goes in the file with the rest of the horse’s records.
But what are we testing for?
A Coggins test is a blood test for equine infectious anemia, commonly shortened to EIA. It is not a general test for anemia, and it won’t tell you whether a horse’s red blood cell count is low. The test looks for antibodies the horse’s immune system has produced against equine infectious anemia virus.
EIA is a viral, bloodborne disease that affects horses, ponies, donkeys, mules and other equids. The virus belongs to the lentivirus group, a family of viruses known for persistent infections. Once a horse contracts EIA, it stays infected for life. No approved vaccine prevents the disease, and no treatment eliminates the virus from an infected horse.
That lifelong carrier status is why the Coggins test matters so much.
Some horses with EIA become seriously sick. Clinical signs can include recurring fever, weakness, weight loss, swelling, depression and anemia. In severe cases, the disease can kill. Many infected horses, however, show few signs or none. A horse can look healthy, perform normally and still carry the virus, putting other horses at risk.
EIA spreads through infected blood. Horse flies and deer flies are important natural carriers. A fly can feed on an infected horse, get interrupted and move to another horse while blood remains on its mouthparts. Contaminated needles, syringes, surgical or dental equipment and anything else that moves blood from one horse to another can also spread the virus.
That’s why controlling EIA takes more than keeping flies away. Use a new sterile needle and syringe for every horse, and clean and disinfect any equipment that could become contaminated with blood.
The name “Coggins test” refers to the agar gel immunodiffusion, or AGID, test developed to detect antibodies to EIA. The test identifies antibodies against an antigen associated with the virus. Today, laboratories may also use approved ELISA tests, which return results faster. Because ELISA tests occasionally produce false positives, labs confirm positive screening results with the AGID, or traditional Coggins, test.
Keep in mind that the test looks for the horse’s immune response to infection rather than the virus itself, so timing can matter. A horse infected very recently may not yet have enough antibodies to test positive. Most infected horses eventually develop detectable antibodies, but there can be a window after exposure when the test comes back negative.
For the average horse owner, the larger purpose of routine testing is surveillance. Testing helps find infected horses before they unknowingly expose other animals.
In Oklahoma, horses attending equine exhibitions such as fairs, shows, rodeos and racetracks must have a negative EIA test from within the previous 12 months. Oklahoma also requires testing when horses mingle in common settings such as boarding and training facilities, as well as before most changes of ownership. Horses entering Oklahoma generally must have a negative EIA test from within the previous 12 months as well.
Those requirements can feel like one more piece of paperwork, especially for owners who travel often and have healthy horses that test negative year after year. But the system works because testing is routine.
There is no vaccine to fall back on if EIA starts spreading through a group of horses, and no cure for the infected horse. A confirmed positive animal remains a carrier for life and must be managed under strict regulatory requirements, generally through euthanasia or permanent isolation from other equids.
So when your veterinarian pulls that tube of blood for another Coggins test, the goal is bigger than getting the paper you need at the gate of the next event. We are looking for evidence of a lifelong, contagious disease that can hide in a healthy-looking horse. Finding those horses before the virus spreads makes that routine blood test one of the simplest and most important tools we have for protecting the horse population.
Equine
A New Set of Challenges
By OKFR Staff
By the time September rolls around in Oklahoma, most of us are more than ready for rain. After weeks or months of dry weather, brown pastures and hard ground, a good soaking rain can seem like the answer to a lot of problems.
For our horses’ feet, the end of a drought can bring a new set of challenges.
During long dry spells, hooves lose moisture and can turn hard and brittle. Dry weather is one of the factors associated with hoof cracks, particularly when combined with long trimming intervals or hoof imbalance. Horses traveling over hard, dry ground also take more concussion, and horses with thin soles or tender feet can grow sore as the ground hardens.
That makes regular farrier care especially important during a drought. You may be tempted to stretch the time between appointments because the feet don’t seem to be growing as fast, but keeping the hoof trimmed and balanced reduces stress on the hoof wall and the structures that support it.
Watch for small cracks and chips before they become big ones. Minor chipping around the bottom of an unshod hoof is fairly routine. A crack that keeps moving upward, gets deeper or causes lameness deserves a closer look from your farrier or veterinarian.
Then comes the rain.
After a long dry spell, hoof moisture can change quickly when horses start standing in wet grass, mud or saturated ground. Going from dry to wet and back again can contribute to brittle hoof walls and cracking.
Wet conditions bring other concerns. Long exposure to moisture can soften the hoof, particularly the sole and frog. Mud and dirty footing favor problems such as thrush and give bacteria more chances to enter through small cracks or separations in the hoof. Hoof abscesses also commonly follow swings between very dry and very wet conditions.
The best defense isn’t complicated. Pick up your horse’s feet regularly and pay attention to what you see and smell. Clean out packed dirt, manure and mud, and watch for cracks, tenderness, a foul odor around the frog or changes in the white line. Give horses a place where they can get out of the mud and stand on clean, dry footing.
Talk with your farrier before trying to change hoof moisture with frequent soaking or a lineup of topical products. More moisture isn’t automatically better. The goal is a healthy, balanced hoof that can adapt to its environment.
Finally, don’t ignore sudden lameness after the weather changes. A hoof abscess can make a horse dramatically lame, sometimes overnight. Many abscesses heal well with proper treatment, but sudden or severe lameness warrants a call to your veterinarian.
We can’t control when Oklahoma’s next rain arrives. Staying consistent with hoof care before, during and after that change gives our horses the best chance of handling it soundly and comfortably.
Equine
More than Just a Saddle Pad
By Savannah Magoteaux
Walk past the horse trailers after a summer jackpot, ranch rodeo or trail ride, and you’ll probably see the same thing: saddle pads draped over gates, fences and trailer doors, drying in the afternoon sun. Some are soaked through with sweat. Others are already stiff with dried salt, caked hair and arena dust.
It’s easy to think of a saddle pad as one more piece of tack, but it has one of the hardest jobs on your horse. On every ride, it cushions the saddle, helps spread pressure across your horse’s back, absorbs sweat and lets heat escape.
A quality saddle pad can last for years if you take care of it.
Built-up sweat, dirt and hair shorten the life of a pad. Over time, they also compress the fibers, reduce the pad’s ability to wick moisture and create stiff spots that can rub your horse’s back. Even the best-made pad can’t do its job once it’s hard as a board.
One of the simplest habits you can develop is cleaning the pad after every ride. Knock off loose dirt, brush away hair and let the pad dry completely before putting it back in the tack room or trailer. Storing a damp pad invites mildew, odors and bacteria and breaks down many materials over time.
Every saddle pad serves the same basic purpose, but the material determines both how it performs and how you should care for it.
For many western riders, wool felt remains the standard. One of its greatest advantages is that it gradually conforms to the horse’s back without losing its structure. To care for a wool felt pad, remove hair with a curry comb or stiff brush after each ride and let it dry thoroughly. When it needs a deeper cleaning, use cool water and a mild wool-safe detergent if the manufacturer recommends one. High-pressure washers, harsh detergents and excessive heat can damage the wool fibers and reduce the pad’s performance.
Traditional woven wool blankets are still popular, particularly beneath ranch saddles or as a top blanket over a felt pad. Shake them out regularly, brush them clean and wash them gently when necessary. Most should air dry, since a dryer can shrink natural fibers.
Neoprene pads have become common because they’re durable, lightweight and easy to clean. Unlike wool, neoprene doesn’t absorb moisture. Many neoprene pads have textured or waffle-patterned surfaces that help move sweat away from the horse while letting air flow beneath the saddle.
You can rinse most neoprene pads with a hose after riding and wash them with mild soap now and then. Let them dry completely before storage, and keep them out of intense heat and direct sunlight, since neoprene can turn brittle over time.
Foam, gel and memory foam inserts show up in many performance pads designed to add shock absorption or relieve pressure points. These materials can cushion well, but many hold more heat than wool. Care varies considerably from one manufacturer to another, so follow the cleaning instructions that came with the pad instead of assuming all synthetic materials wash the same way.
Synthetic fleece pads are another common option, particularly for everyday riding. They are generally easier to clean than natural wool, and many can go through a washing machine on a gentle cycle before air drying. Regular brushing between washings keeps the fleece from matting and packing with hair.
Whatever type of saddle pad you choose, no pad can make up for a saddle that doesn’t fit. Even the thickest pad cannot eliminate pressure points created by a poorly fitting saddle. Think of the pad as a partner to proper saddle fit, helping protect your horse while the saddle does its job.
Before your next ride, flip your saddle pad over and run your hand across the side that rests against your horse. If it feels rough, stiff or crusted with dried sweat and hair, imagine how it feels after an hour in the saddle.
A few minutes spent cleaning and drying your pad after each ride will help it last longer, protect your investment and keep your horse more comfortable every time you swing a leg over.
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