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Equine

Core Vaccines Every Horse Needs

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There are many possible dreadful diseases that horses can contract through various insect vectors, ticks, wildlife and even wounds that are rather easy to prevent with routine core vaccinations. Oklahoma has just about every insect known to man to offer, as all Oklahomans are fully aware of especially in the spring and summer, making it a very good environment for some of these key viruses to spread. The five types of diseases that will be discussed in this article are commonly referred to as the deadly five, and that is why they make the core vaccine list because if one of these diseases is contracted it can be game over for your horse. Before spring arrives, the core vaccines should be on every horse owners to do list as a common health care checklist that should be just as routine as deworming and dental care.

The American Association of Equine Practitioners (AAEP) and American Veterinary Medical Association (AVMA) have established key guidelines for vaccinations for horses that are required based on risk to the public and to the horse themselves. The five diseases listed as core vaccines are Rabies, Tetanus, Eastern equine encephalomyelitis virus, Western equine encephalomyelitis virus and West Nile virus.

The AVMA defines core vaccinations as those “that protect from diseases that are endemic to a region, those with potential public health significance, required by law, virulent/highly infectious, and/or those posing a risk of severe disease. Core vaccines have clearly demonstrated efficacy and safety, and thus exhibit a high enough level of patient benefit and low enough level of risk to justify their use in the majority of patients.”  (AAEP website)

The reason these deadly five diseases are listed as core vaccines is because they pose a possible public health risk, are highly virulent or infectious, have a high mortality rate and can be effectively controlled with routine vaccination with great efficacy. It’s important to remember that vaccines are not 100% effective for every disease but well worth that risk when it comes to protect against these five deadly diseases.

There are many other diseases that horses may or may not need to be protected against with vaccinations. These diseases are listed under “risk based” immunizations depending on travel, environment, geographic circumstances and even competition/racing venue requirements before entering into an event. With recent localized outbreaks of equine herpes virus at race tracks, shows and barrel races, many venues and race tracks require very recent vaccinations for these viruses before permitted onto the grounds. Here is a list of risked based diseases vaccinations are available for: Anthrax, botulism, equine herpes virus 1 & 4, equine viral arteritis, influenza, rotaviral diarrhea and strangles. These diseases and the risk of contracting them must be weighed on an individual horse bases versus the risk of the vaccine itself.

Many vaccines today that are available for horses have all core vaccines in one shot except rabies. One vaccine provider has developed the first all five core diseases in one single vaccine. Horses just like other species require multiple boosters to generate an appropriate immune response to be protective. Unvaccinated horses or horses that don’t have a documented vaccine history it is recommended to have at least one booster four to six weeks after the first dose to get an adequate response and then annually after that. In some endemic areas of the country with higher cases of West Nile and other viral encephalomyelitis diseases due to prolonged mosquito vector season it is often recommended to have a booster in the mid-to-late summer to have added protection against these particular viruses.

Below is a breakdown of each disease and its importance for your horse and even yourself to be protected against.

Rabies –

Highly deadly virus that is unsurvivable for any mammal that contracts the disease and begins to show clinical signs, even for humans. As of 2016 only four people have survived rabies infection after showing clinical signs, so the odds are very poor. One of the most difficult aspects of rabies in horses is the variability of clinical signs. Horses can display two clinical manifestations of the disease: Furious/Mad form or Dumb/Stuporous but really it can look like anything early on. Veterinarians are trained that rabies can display almost as anything and should always be on the list for a horse that presents with neurologic symptoms, especially horses with no vaccine history for rabies. Horses can display colic, ataxia, blindness, hypersalivation, depression, weakness and even lameness. Also there is no ante-mortem test meaning there is no test to confirm rabies until the horse is dead or euthanized and the brain is collected for testing. Rabies is transmitted via bites from infected rabid wildlife such as skunks (highest transmitter in Oklahoma), raccoons, fox and bats.

EEE/WEE –

Eastern and Western equine encephalomyelitis is transmitted like other viral enchephalitides via the mosquito vector. Clinical signs are neurologic signs of ataxia, blindness, head pressing or head tilt, recumbency, seizures and paralysis of the throat/tongue. Only up to 15% of horses survive EEE. WEE has a better survival rate up to 50% and has very similar sign as EEE. Birds are common carriers of EEE/WEE and are the reservoir for these viruses.

West Nile –

A very commonly know virus that is also transmitted by mosquitoes to horses, humans, birds and other mammals. Birds again are the largest reservoir for this virus. The virus affects the central nervous system by creating inflammation around the brain and spinal cord. Symptoms or clinical signs are fever, depression, muscle fasciculations, weakness and ataxia. The severity of the disease is horse dependent and not all horses get the disease but among those horses that do about 1/3 will die from it. Also almost half of horses will have lasting neurologic effects from the disease.

Tetanus –

The causative bacterium was first discovered in 1884 by injecting wound exudates from humans with tetanus into animals leading to the discovery that Clostridium tetani is the causative bacteria. The bacteria is found everywhere in the environment including the soil and manure of horses. Horses are rather sensitive to the neurotoxin produced from Clostridium tetani that leads to the clinical signs of tetanus. The bacteria enter the horse’s body through wounds, cuts or even surgical wounds such as castration sites. The spores of the bacteria turn into the vegetative form when it begins to grow and replicate. The neurotoxin produced from the bacteria enters the central nervous system by peripheral nerves. The clinical signs of tetanus are muscle spasms, stiffness, prolapsed third eyelids, extended head and neck, elevated tail and retracted lips. As the disease progresses, horses will become recumbent and have difficulty breathing. Life saving treatments can be implemented to save the infected horses but still up to 75% of horses will die.

What all these disease have in common besides death is that these deaths are extremely excruciating, painful and miserable. There is no doubt from seeing these cases personally and from watching teaching videos that these horses are suffering enormously. What makes it even more difficult and disheartening when we see these cases is that they are so easily preventable with vaccination. The bottom line is that vaccines can save your horse’s life from these deadly diseases and vaccines are rather inexpensive especially compared to the expensive of trying to treat the horse once they are ill. Please talk to your veterinarian about getting your horse or horses protected with core vaccinations and if any other vaccinations are necessary based on your situation.

Read more great articles in the March 2020 issue of Oklahoma Farm & Ranch.

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Equine

Gastric Ulcers

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By Dr. Devan England DVM

Does your horse have gastric ulcers? Gastric or stomach ulcers are frequently blamed for a variety of things including poor performance, acting ‘cinchy’, weight loss, not eating, poor coat condition, diarrhea and colic. However, gastric ulcers are not always the culprit and the only way to know for sure if your horse has gastric ulcers is to look at the stomach on camera, using an endoscope. Poor appetite and poor body condition are the mostly widely observed clinical signs with gastric ulcers, however, these are non-specific. If you think your horse might have gastric ulcers, the best place to start is to talk to your veterinarian and consider scheduling a gastroscopy. Gastroscopy requires the horse be held off feed for at least 16-18 hours and held off water for at least 6-8 hours. Fasting off feed and water is necessary to allow the veterinarian to see the whole stomach. If restricting feed or water is difficult in your management situation, many veterinarians will allow you to hospitalize your horse the night before gastroscopy for proper fasting.

Gastric ulcers are split into two types, classified by the location of the ulcer in the stomach. Squamous ulcers are ulcers that occur in the squamous or skin like portion of the stomach. This is the top part of the horse’s stomach, is closest to the esophagus, and has squamous tissue to protect this portion of the stomach from stomach acids. The other ulcer type are glandular ulcers. Glandular ulcers occur in the bottom portion of the stomach, which is closest to the small intestine. This portion of the stomach has glandular mucosa with cells responsible for producing stomach acids for digestion as well as cells that produce mucus and buffers to protect the lining from stomach acid. Gastroscopy is important not only for diagnosing whether ulcers are present but also determining the severity and the type of ulcer, because these two ulcer types require different treatments.

Squamous gastric ulcers are common in racehorses both in and out of training, with higher prevalence in racehorses under training. Prevalence in Thoroughbred racehorses in training has been reported to be up to 100% (Sykes 2015). Squamous ulcers are also prevalent in Western pleasure horses, Thoroughbred stallions on breeding farms, and Italian donkeys (Sykes 2015). Glandular gastric ulcer prevalence has not been as well described as squamous ulcers. Glandular ulcers are reported to be most common in Thoroughbred and Standardbred racehorses, Canadian showjumpers and polo ponies, and American Quarter Horses (Sykes 2015).

Risk factors for ulcers vary by ulcer type. Anti-inflammatories (Bute, Banamine) can increase the risk of glandular ulcers in some horses by affecting normal defense mechanisms but are not a high risk in most horses. Horses that display stereotypic behaviors, such as cribbing, have an increased risk of squamous ulcers. Grain fed before hay in non-exercising horses, feeding larger amounts of grain, and increased time between meals increases the risk of squamous ulcers. Increased time with high intensity exercise and housing in single pens is associated with increased risk of glandular ulcers. A straw only diet, lack of water access and lack of direct contact with other horses increases the general risk of gastric ulcers.

If your horse is diagnosed with ulcers, the mainstay of treatment is a buffered formulation of omeprazole (Gastrogard, Ulcergard). Over the counter Omeprazole and compounded Omeprazole are not effective because without buffering, the acidic stomach quickly breaks down the drug before absorption. Most horses with squamous ulcers will have healing of these ulcers after a 4-week course of Gastrogard or Ulcergard at treatment dose (whole tube for the average horse). Some horses may be healed by 3 weeks of treatment, but all horses should undergo a recheck gastroscopy before stopping treatment. Horses diagnosed with glandular ulcers need combination therapy with Gastrogard/Ulcergard and Sucralfate for 4 weeks. About 2/3 of horses with glandular ulcers will heal in this time, but some horses may require longer treatment times so a recheck is always recommended before discontinuing treatment.

Horses at higher risk of gastric ulcers may benefit from preventative (low) doses of Ulcergard (1/4 tube in average sized horse) given for a few days before and during high stress situations like long distance travel and competitions. Sea buckthorn berry supplement may be protective against formation of glandular ulcers. Dietary management to decrease the risk of ulcers includes providing more frequent small hay meals if pasture access is not available, limiting high sugar grains as much as possible and adding vegetable oil to the feed.

Sykes BW, Hewetson M, Hepburn RJ, Luthersson N, Tamzali Y. European college of equine internal medicine consensus statement – equine gastric ulcer syndrome in adult horses. J Vet Internal Med 2015; 29:1288-1299.

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Equine

Breeding the Older Mare

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By the time the June issue reaches readers, the regular breeding season is winding down for many horse owners. Some mares are already confirmed in foal, some breeding decisions have been pushed aside for another year and some owners are already thinking ahead to next season. For those with an older mare they hope to breed, that early planning can make a real difference.

Older mares can still produce foals, but they may need more help than younger mares. Age affects the reproductive system, and those changes can make it harder for a mare to become pregnant, stay pregnant or carry a healthy foal to term. That does not mean every older mare is a poor candidate for breeding. It does mean owners should go into the process with realistic expectations and a good veterinarian involved from the beginning.

A mare’s reproductive age does not always match her number of birthdays. Some mares in their mid-to-late teens settle easily and carry foals without much trouble. Others begin having problems earlier. Past reproductive history matters. A mare that has had regular foals may be different from an older maiden mare that spent most of her life showing, racing, working or sitting open. Health, body condition, uterine health and conformation all play a role.

One of the main challenges with older mares is egg quality. As mares age, their eggs age, too. Older eggs are more likely to have abnormalities that prevent fertilization, stop early embryo development or lead to early pregnancy loss. Even when breeding is timed well and the stallion has good fertility, the mare may not settle because the egg itself is no longer as viable as it once was.

The uterus also changes with age. The lining of the uterus, called the endometrium, can become less healthy and less able to support pregnancy. Over time, some mares develop fibrosis or scarring within the uterus. This can interfere with the placenta’s ability to develop and support a growing fetus. A mare may get pregnant early, but then lose the pregnancy because the uterus cannot maintain it.

Older maiden mares can face an additional problem. When a mare cycles, it is normal for some fluid to be present in the uterus. A healthy reproductive tract clears that fluid through uterine contractions and cervical relaxation. In some older mares, especially those that have never had a foal, the cervix may not relax well enough. Fluid can build up and remain in the uterus after breeding. That fluid can damage sperm, interfere with an embryo and increase the risk of infection or inflammation.

Post-breeding inflammation is another concern. All mares experience some uterine inflammation after breeding, but most clear it within a reasonable amount of time. Older mares may not clear it well. This can leave the uterus in a poor environment for pregnancy. A veterinarian may recommend uterine lavage, medication or other treatment after breeding to help the mare clear fluid and inflammation.

External conformation can also affect fertility. As some mares age, especially taller, thinner mares or mares that have had multiple foals, the reproductive tract may tilt in a way that allows manure or air to contaminate the vulva and vagina. Poor vulvar conformation can increase the risk of uterine infection. In some cases, a veterinarian may recommend a Caslick’s procedure to help protect the reproductive tract.

Because so many factors can be involved, an older mare should have a breeding soundness exam before the next season begins. This may include a physical exam, reproductive ultrasound, uterine culture, cytology and possibly a uterine biopsy. These tools help determine whether the mare has infection, inflammation, fluid retention, poor uterine health or other issues that need to be addressed before breeding.

Timing is also important. With an older mare, it is usually better to start early in the season rather than waiting until late. More cycles give the veterinarian more chances to manage the mare properly. Early planning also allows time to treat infection, improve body condition, schedule semen shipments, evaluate the stallion’s fertility and consider whether cooled semen, frozen semen, live cover or another option makes the most sense.

Owners should also look closely at the mare’s overall health. A mare that is too thin, too heavy, metabolically unstable, lame or dealing with chronic illness may struggle to conceive or carry a foal. Good nutrition, dental care, hoof care and vaccination planning all matter. Breeding may be a reproductive decision, but pregnancy affects the whole horse.

For some mares, advanced reproductive options may be worth discussing. Embryo transfer allows a valuable mare to produce a foal without carrying it herself. This may be helpful for older mares that can produce an embryo but should not carry a pregnancy, or for mares that are still competing. Other assisted reproductive techniques may also be available through specialized equine reproduction centers.

The hard truth is that breeding an older mare can take more time, money and patience. Not every mare will get in foal, even with good care. Some mares should not be bred if the risks are too high. Still, many older mares can produce healthy foals when problems are identified early and managed correctly.

For owners hoping to raise one more foal from a favorite mare, the best plan is to start the conversation before breeding season arrives. Talk with a veterinarian, evaluate the mare honestly and make decisions based on her health, history and reproductive exam. Hope is part of breeding horses, but planning gives that hope a much better chance.

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Equine

Here’s to the Great Horses

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By Summer McMillen

Everyone that knows anything about horses knows that there are bad ones, good ones, and great ones.

The bad ones are good for nothing. You can’t catch them, you can’t saddle them, and you can’t get on them without feeling like you need a helmet, some kind of padded vest and, an instruction manual. Once you do mount up the whole ride is a battle and heaven forbid, you actually have a job to do because they are little to no help in holding the herd. We all find ourselves owning a bad one or two throughout our lives. Best case scenario is they find a more tolerable home to go to through via a horse sale or the classifieds. Worst case scenario all you can do is say “Vaya con Dios,” put a sign on them that reads “Do Not Attempt,” and turn them out to pasture. Hoping they are decent enough to stay within the borders and make a beautiful yard ornament.

Good horses are usually much more tolerable. They’re pretty easy to catch, saddle, and hop up on. Sometimes they might have a bad habit or two like setting back when they’re tied to a fence or, getting cold backed on early mornings that you tolerate because they are so skilled in a specific field. A good horse is usually only good for one thing. They have a niche talent m, if you will. They can be a good heel horse. A good head horse. The horse you want to gather pastures on because you know he won’t knicker or rare up when you get dropped off in the jig line. A good kid horse. Your rodeo horse. The horse you put your wife on when she’s being a little wimpy that day. Good horses usually get sold because they are proficient in their given field and they find good homes making both parties happy. We will all own many good horses in our lives and be happy to do so.

Great horses are a rare and treasured possession. They are simultaneously easy and hard to own. Easy because you can do anything on them. Hard because everyone is always trying to buy them from you. A great horse stands still while your kid pulls their head down all the way to the ground so they can halter them. A great horse is never cold backed and always ready to cinch tight and take off. A great horse can be ridden in the pasture and the rodeo arena on the same day. A great horse doesn’t need practice. A great horse is always willing to do anything you ask of them at any given moment. Great horses find their homes as horse colts and usually live out the rest of their days at the same home because great horses are irreplaceable.

People and horses are not all that different. There are bad, good, and great ones. The more time I spend around horses the more I am convinced of the kind of person I want to be. “Bad” will absolutely not do. “Good“ is much too common  and just doesn’t quite cut it more often than not. “Great” is what I aspire to be.

Great can be defined in so many ways when we let human standards get involved but, I want to be great as defined in the qualities of a great horses.

I want to be kind and patient while my children are learning. I want to be ready to help anyone who asks me. I want to go the extra mile. I want to make my home a beautiful place to come to after a full day’s work outside. I want to not be thrown off by life’s twists and turns but, firm in my faith.

So, basically what I’m saying is I want to be a great horse. And honestly there are worse things we could all aspire to be.

Here’s to great horses. May we know them, love them, and if we’re lucky be great just like them.

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